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Polaris ATV pictures | 2012 Ranger Crew 500 | one of motorbike parts details. Here motorcycles and motor bike information, happy to read Polaris ATV pictures | 2012 Ranger Crew 500 | information.
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Health and Fitness | Health and Fitness | * Written by Randy Reisman | Wednesday, 01 August 2012 04:23 | Word Count: 582
This past June 2012 was Migraine Awareness Month and for the over 30 million Americans that suffer from migraines, awareness is just one of the ways to draw attention to this debilitating affliction. If you suffer from chronic migraine headaches you don't need to be made aware of the throbbing pain, nausea, vomiting, dizziness, and sensitivity to almost every sense that comes with every attack. However, there are a few good reasons to champion the cause behind Migraine Awareness Month for everyone, not just the millions of Americans that live with the effects of these unbearable neurological nightmares.
Two of the biggest reasons behind Migraine Awareness month are in trying to rid ourselves of the myths and misunderstandings that result in stigmatizing those suffering from migraine headaches. The other is in trying to make migraines more visible to the public in general and to try to raise more money for research, which with a little luck and hope will lead to better treatment options.
From all accounts Migraine Awareness Month was successful in achieving its stated goals, at least as a starting point. By expanding their efforts to get the word out to a larger audience about migraine headaches, the National Headache Foundation, sponsors of Migraine Awareness Month, reports over 90 million media exposures, which is a measure of the number of times the message of the cause was presented to readers, viewers, or listeners to public media.
In addition to drawing more attention to the condition, organizers also hope to provide education and information to migraine sufferers about treatment options such as preventative therapies. Not all migraines are alike with some people experiencing just an occasional attack while others have several attacks per month, the result of which is a life spent on the coach, in bed, or simply waiting for the next migraine to put you there. The National Headache Foundation reports that one in four households in America has a family member suffering from migraine headaches. Migraines are three times more common in women than men, affecting 30% of all women over a lifetime. And there is a dollars and cents side to this health problem with migraines said to cost the U.S. more than $20 billion every year, including direct medical expenses such as doctor visits and medications as well as indirect expenses including missed time at work and lost productivity. The World Health Organization notes that migraine headaches are one of the 20 most debilitating medical illnesses on earth today.
The National Headache Foundation states that their mission is to help change the lives of those experiencing headaches and some of the many services that they offer to strengthen awareness of migraine headaches include HeadWise magazine, which is the first national magazine for and about people with migraine and other headache disorders. They have also developed other techniques for raising awareness, bringing people together and raising funds to fight migraine headaches with initiatives such as NHF Chat rooms, NHF podcasts, NHF Awareness Ribbons, and other events such as public education, research, and Migraine Masterpieces, an art contest that provides an outlet for migraine sufferers of all ages to express their artistic abilities.
For their part the founders of Migraine Awareness Month, the National Headache Foundation, see this event as part of their on-going efforts to raise the public consciousness and awareness of migraine headaches as well as raising resources necessary to combat this affliction. With so many Americans as well as those sufferers around the world having their daily lives disrupted by the symptoms and effects of migraine headaches it's not a surprise that this event was, by all accounts, a tremendous success and one that is sure to help the cause going forward.
To know more about Migraine Relief please browse http://www.migramedic.com/">Migraine Relief
Mars mission will probe Red Planet?s past, present
Web edition : 5:12 pm
Editor?s note:This is the second of two articles previewing the Mars Curiosity rover?s upcoming Mars landing. (See also "Curiosity readies for dramatic entrance.") The vehicle is scheduled to land on Sunday evening, August 5, Pacific Daylight Time. Science News astronomy writer Nadia Drake will cover the landing live from NASA?s Jet Propulsion Laboratory in Pasadena, Calif.
If, late in the evening of August 5, NASA?s Curiosity rover survives what might be the most daring interplanetary touchdown in history, the six-wheeled robot will find itself in a dramatic landscape ripe with research opportunities: Gale Crater, an enormous basin with a 5-kilometer-tall mountain in the middle, called Mount Sharp. There, Curiosity will look for evidence of water, energy sources and organic carbon ? the hallmarks of life-friendly environments, past or present.
By reading the rocky clues hidden in that mountain, the rover will also try to peer back in time and learn what environments on ancient Mars were like. For 98 weeks ? one Mars year ? the rover and its 75 kilograms of science instruments will attempt to probe the deep Martian past.
Though the mountain is the mission?s primary target, scientists hope the rover will first alight on one particularly intriguing patch of rock.
?We don?t really know what that material is,? says project scientist John Grotzinger of Caltech.
Unlike the rest of the crater floor, this rock appears to be hard and capable of holding onto the heat of the day. In the absence of volcanic activity, that property is suggestive of compounds cemented together by water. Since scientists are interested in studying where water flowed on ancient Mars, the warm, rocky patch would be an ideal place for the rover to start poking around.
But not for too long. Mount Sharp is ?going to be there on the horizon, calling to us,? says Ryan Anderson of the U.S. Geological Survey Astrogeology Science Center in Flagstaff, Ariz.
The ancient pile of sediments, rising higher above the surrounding terrain than any mountain in the Lower 48, holds in it the rocky clues to understanding ancient Martian environments and past habitability. The evidence takes the form of mineral layers, some of which ? like the clays near the mountain?s base ? require water to form. As the rover climbs Mount Sharp, the clues it deciphers will help scientists understand how a planet that was once more like Earth became the dry, dusty and acidic ball it is today. ?Suddenly, the planet seems to have gotten dry,? Grotzinger says. ?I like to call it the great desiccation event.?
The rover will analyze rocks with a variety of instruments. One, the ChemCam, uses a laser to vaporize a small sample of rock or soil from up to seven meters away. Then, scientists can study the ingredients. Another instrument will drill into rocks, creating a powder that the rover can ingest and analyze for signs of things like organic carbon ? a job that Grotzinger says is probably the most difficult the rover will encounter. ?This is a really hard thing to do, even on Earth,? he says. ?Even on a planet that?s teeming with life, [organic carbon] almost never gets preserved.?
Comparing Mars and Earth may help scientists understand how environmental triggers influence the evolution of life. When studying life?s origins and diversity on Earth, Grotzinger says, scientists often wonder what would have happened if specific events ? such as the flooding of Earth?s atmosphere with oxygen about 2.4 billion years ago ? hadn?t occurred. ?Is there some place that you can compare to where that didn?t happen?? That?s Mars,? he says.
Protein involved in DNA replication, centrosome regulation linked to dwarfism, small brain sizePublic release date: 31-Jul-2012 [ | E-mail | Share ]
Contact: Peter Tarr tarr@cshl.edu 516-367-8455 Cold Spring Harbor Laboratory
Orc1 mutations may explain pathology in manifestations of Meier-Gorlin syndrome
Cold Spring Harbor, NY Research published Aug. 1 by scientists at Cold Spring Harbor Laboratory (CSHL) links gene mutations found in some patients with Meier-Gorlin syndrome (MGS) with specific cellular dysfunctions that are thought to give rise to a particularly extreme version of dwarfism, small brain size, and other manifestations of abnormal growth which generally characterize that rare condition.
Although only 53 cases of Meier-Gorlin syndrome have been reported in the medical literature since the first patient was described in 1959, it is a malady whose mechanisms are bringing to light new functions for some of the cellular processes common to all life. Pathology related to MGS is traced in the new research to one of these, the fundamental process called mitosis in which cells replicate their genetic material and prepare to divide into two identical "daughter" cells.
CSHL President and Professor Bruce Stillman, Ph.D., a cancer biologist who has made seminal discoveries over three decades that have helped reveal the exquisite choreography of how chromosomes are duplicated in cells, led the new research, which suggests how, during mitosis, mutant versions of a protein called Orc1 contribute in two distinct ways to severe MGS pathology. The research is published online ahead of print in Genes & Development.
Components of the cellular replication machinery
Orc1 is the largest of six proteins that form parts of a cellular machine called the origin recognition complex, or ORC. As Stillman and others discovered 20 years ago, ORC in human cells attaches to DNA at specific locations throughout the genome when a cell is preparing to duplicate its genetic information and go through cell division. These locations are called replication start sites or origins. When ORC and several other helper proteins attach at these positions, each of the assembled groups of proteins is said to form a pre-replication complex (pre-RC).
When pre-RCs have attached at all of the "start" sites throughout the genome -- a process called licensing -- cells can then begin to copy the double helix of DNA to produce two copies, one for each daughter cell. ORC enables a cell to keep track of its DNA replication and this is essential because the genetic material must be copied once, and only once.
Other roles for ORC, including centrosome regulation
Over the years, new roles for many of the ORC proteins have been found within the cell. Not only are they involved in DNA replication; as Stillman (2009) and others have shown, a number of them are also known to be involved in regulation of a cellular organ called the centrosome. Floating inside the cell's watery cytoplasm, the centrosome helps regulate the cell's progression through the cell-cycle. It ensures that the duplicated chromosomes are segregated evenly to the daughter cells. During replication, it organizes the threadlike microtubules that form a delicate spindle that segregates the two identical sets of chromosomes. Serving as anchor points for those "threads" are tiny structures inside the centrosome called centrioles. Just like DNA, which must be licensed to insure it is only copied once per cell cycle, so must the centrosome and centrioles be licensed for proper regulation of cell division.
The centrosome also plays a role in establishing cell outgrowths, such as axons in brain cells and cilia in many other cells of the body.
Mutant ORC proteins are linked to MGS pathology
Stillman and Manzar Hossain, Ph.D., a postdoctoral student in the Stillman lab, have followed up clues in recent research linking mutations in the genes encoding proteins of the pre-RC, including Orc1, with mutations found in Meier-Gorlin syndrome patients. In a study published this past February, a multinational research team noted the relationship of such mutations to various manifestations of the pathology seen in 35 MGS patients. They noted that MGS patients with mutations in Orc1 were the shortest and had the smallest brain size.
The new study by Hossain and Stillman describes how Orc1 mutations cause cellular dysfunctions that contribute directly to the most severe instances of dwarfism and small brain size.
New findings about centrosome and centriole dysfunction in MGS
In their dissection of Orc1, Hossain and Stillman discovered that different domains of the Orc1 protein control centrosome copy number and DNA replication; and that both are involved in MGS pathology.
The CSHL scientists suggest that centrosome reduplication as well as dysregulation of DNA replication (in which Orc1 is also involved) are directly associated with the more severe manifestation of dwarfism and microcephaly, or abnormally small brain size, seen in the most pronounced MGS cases.
For the first time, they observed that when the Orc1-encoding gene is mutated in a manner observed in MGS patients, the role normally played by the Orc1 protein in preventing the centrosome from re-duplicating itself is disturbed. By causing defects in the duplication of centrosomes, processes such as cell division and cell signaling can go awry, leading to the severe growth defects found in Orc1 MGS patients.
This research has significant implications for processes that control body and brain size; how tissues come to be the size that they are is a little understood area of biology. Studying how mutations in centrosome biology affect tissue and body size may lead to insights into this regulation, Stillman suggests. He also notes that while Orc1 MGS individuals have a relatively small brain, they display normal intelligence, suggesting the enormous potential of the human brain.
###
"Meier-Gorlin syndrome mutations disrupt an Orc1 CDK inhibitory domain and cause centrosome reduplication" appears online a head of print on August 14, 2012 in Genes & Development. The authors are: Manzar Hossain and Bruce Stillman. The paper can be viewed online at: http://genesdev.cshlp.org
This research was supported by a grant from the National Cancer Institute [CA13106].
About Cold Spring Harbor Laboratory
Founded in 1890, Cold Spring Harbor Laboratory (CSHL) has shaped contemporary biomedical research and education with programs in cancer, neuroscience, plant biology and quantitative biology. CSHL is ranked number one in the world by Thomson Reuters for impact of its research in molecular biology and genetics. The Laboratory has been home to eight Nobel Prize winners. Today, CSHL's multidisciplinary scientific community is more than 360 scientists strong and its Meetings & Courses program hosts more than 12,500 scientists from around the world each year to its Long Island campus and its China center. Tens of thousands more benefit from the research, reviews, and ideas published in journals and books distributed internationally by CSHL Press. The Laboratory's education arm also includes a graduate school and programs for undergraduates as well as middle and high school students and teachers. CSHL is a private, not-for-profit institution on the north shore of Long Island. For more information, visit www.cshl.edu.
[ | E-mail | Share ]
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AAAS and EurekAlert! are not responsible for the accuracy of news releases posted to EurekAlert! by contributing institutions or for the use of any information through the EurekAlert! system.
Protein involved in DNA replication, centrosome regulation linked to dwarfism, small brain sizePublic release date: 31-Jul-2012 [ | E-mail | Share ]
Contact: Peter Tarr tarr@cshl.edu 516-367-8455 Cold Spring Harbor Laboratory
Orc1 mutations may explain pathology in manifestations of Meier-Gorlin syndrome
Cold Spring Harbor, NY Research published Aug. 1 by scientists at Cold Spring Harbor Laboratory (CSHL) links gene mutations found in some patients with Meier-Gorlin syndrome (MGS) with specific cellular dysfunctions that are thought to give rise to a particularly extreme version of dwarfism, small brain size, and other manifestations of abnormal growth which generally characterize that rare condition.
Although only 53 cases of Meier-Gorlin syndrome have been reported in the medical literature since the first patient was described in 1959, it is a malady whose mechanisms are bringing to light new functions for some of the cellular processes common to all life. Pathology related to MGS is traced in the new research to one of these, the fundamental process called mitosis in which cells replicate their genetic material and prepare to divide into two identical "daughter" cells.
CSHL President and Professor Bruce Stillman, Ph.D., a cancer biologist who has made seminal discoveries over three decades that have helped reveal the exquisite choreography of how chromosomes are duplicated in cells, led the new research, which suggests how, during mitosis, mutant versions of a protein called Orc1 contribute in two distinct ways to severe MGS pathology. The research is published online ahead of print in Genes & Development.
Components of the cellular replication machinery
Orc1 is the largest of six proteins that form parts of a cellular machine called the origin recognition complex, or ORC. As Stillman and others discovered 20 years ago, ORC in human cells attaches to DNA at specific locations throughout the genome when a cell is preparing to duplicate its genetic information and go through cell division. These locations are called replication start sites or origins. When ORC and several other helper proteins attach at these positions, each of the assembled groups of proteins is said to form a pre-replication complex (pre-RC).
When pre-RCs have attached at all of the "start" sites throughout the genome -- a process called licensing -- cells can then begin to copy the double helix of DNA to produce two copies, one for each daughter cell. ORC enables a cell to keep track of its DNA replication and this is essential because the genetic material must be copied once, and only once.
Other roles for ORC, including centrosome regulation
Over the years, new roles for many of the ORC proteins have been found within the cell. Not only are they involved in DNA replication; as Stillman (2009) and others have shown, a number of them are also known to be involved in regulation of a cellular organ called the centrosome. Floating inside the cell's watery cytoplasm, the centrosome helps regulate the cell's progression through the cell-cycle. It ensures that the duplicated chromosomes are segregated evenly to the daughter cells. During replication, it organizes the threadlike microtubules that form a delicate spindle that segregates the two identical sets of chromosomes. Serving as anchor points for those "threads" are tiny structures inside the centrosome called centrioles. Just like DNA, which must be licensed to insure it is only copied once per cell cycle, so must the centrosome and centrioles be licensed for proper regulation of cell division.
The centrosome also plays a role in establishing cell outgrowths, such as axons in brain cells and cilia in many other cells of the body.
Mutant ORC proteins are linked to MGS pathology
Stillman and Manzar Hossain, Ph.D., a postdoctoral student in the Stillman lab, have followed up clues in recent research linking mutations in the genes encoding proteins of the pre-RC, including Orc1, with mutations found in Meier-Gorlin syndrome patients. In a study published this past February, a multinational research team noted the relationship of such mutations to various manifestations of the pathology seen in 35 MGS patients. They noted that MGS patients with mutations in Orc1 were the shortest and had the smallest brain size.
The new study by Hossain and Stillman describes how Orc1 mutations cause cellular dysfunctions that contribute directly to the most severe instances of dwarfism and small brain size.
New findings about centrosome and centriole dysfunction in MGS
In their dissection of Orc1, Hossain and Stillman discovered that different domains of the Orc1 protein control centrosome copy number and DNA replication; and that both are involved in MGS pathology.
The CSHL scientists suggest that centrosome reduplication as well as dysregulation of DNA replication (in which Orc1 is also involved) are directly associated with the more severe manifestation of dwarfism and microcephaly, or abnormally small brain size, seen in the most pronounced MGS cases.
For the first time, they observed that when the Orc1-encoding gene is mutated in a manner observed in MGS patients, the role normally played by the Orc1 protein in preventing the centrosome from re-duplicating itself is disturbed. By causing defects in the duplication of centrosomes, processes such as cell division and cell signaling can go awry, leading to the severe growth defects found in Orc1 MGS patients.
This research has significant implications for processes that control body and brain size; how tissues come to be the size that they are is a little understood area of biology. Studying how mutations in centrosome biology affect tissue and body size may lead to insights into this regulation, Stillman suggests. He also notes that while Orc1 MGS individuals have a relatively small brain, they display normal intelligence, suggesting the enormous potential of the human brain.
###
"Meier-Gorlin syndrome mutations disrupt an Orc1 CDK inhibitory domain and cause centrosome reduplication" appears online a head of print on August 14, 2012 in Genes & Development. The authors are: Manzar Hossain and Bruce Stillman. The paper can be viewed online at: http://genesdev.cshlp.org
This research was supported by a grant from the National Cancer Institute [CA13106].
About Cold Spring Harbor Laboratory
Founded in 1890, Cold Spring Harbor Laboratory (CSHL) has shaped contemporary biomedical research and education with programs in cancer, neuroscience, plant biology and quantitative biology. CSHL is ranked number one in the world by Thomson Reuters for impact of its research in molecular biology and genetics. The Laboratory has been home to eight Nobel Prize winners. Today, CSHL's multidisciplinary scientific community is more than 360 scientists strong and its Meetings & Courses program hosts more than 12,500 scientists from around the world each year to its Long Island campus and its China center. Tens of thousands more benefit from the research, reviews, and ideas published in journals and books distributed internationally by CSHL Press. The Laboratory's education arm also includes a graduate school and programs for undergraduates as well as middle and high school students and teachers. CSHL is a private, not-for-profit institution on the north shore of Long Island. For more information, visit www.cshl.edu.
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?
AAAS and EurekAlert! are not responsible for the accuracy of news releases posted to EurekAlert! by contributing institutions or for the use of any information through the EurekAlert! system.
Whenever I can steal time with my kids and grandkids I?ll take it, so last week those of us who live locally escaped to Big Bear Lake for a few days. I brought home some fantastic memories, and one great insight.
It was a wonderful time of boating and parasailing on the lake, roughhousing with my three grandsons, and playing board games with my family and laughing so hard it hurt. On the last morning we decided to take a walk before wrapping things up.
The neighborhood was gorgeous. Luxurious vacation rental homes were surrounded dark green pine trees and framed by that uniquely mountain blue sky. My son, Derek, watched the unusual way my three-year-old grandson, Ryder, was walking.
?
?What are you doing?? he asked Ryder. Ryder had been looking down as he walked close to his Aunt Bethany, carefully moving, stopping, or turning when she did. He had stayed close to her side most of the weekend, so this was no exception. He shyly looked up at Derek, and very softly confessed: ?I like to walk in her shadow.?
He chose to walk in her shadow. Because he loves her and enjoyed her company, he thought it a good thing to copy what she did.
And there is was?I realized that all I really want is to walk in Jesus? shadow.
If I am in His shadow, I am also surrounded and enfolded by His presence. I am near to Him, following Him, and walking where He leads. And there is no place I would rather be.
Like Ryder, I choose to walk in the shadow of the One I love.
How priceless is your unfailing love, O God! People take refuge in the shadow of your wings.???????????????????????????????????????????????????????????????????????????????????????????????????????????????????????????? Psalm 36:6-7
Whoever dwells in the shelter of the Most High will rest in the shadow of the Almighty.I will say of the Lord, ?He is my refuge and my fortress, my God, in whom I trust.?????????????????????????????????????????????????????????????????????????????????????????????????????????????????????????????? Psalm 91:1-2
Further reflection:
Does walking in someone?s shadow have a positive or negative connotation for you?
Take your journal (or other paper) and make a list of the benefits of walking in Jesus? shadow.
Lord, I want to walk in Your shadow. I yearn to stay close to You, to go where You go, and follow where You lead. Thank You for making room for me in the shadow of your wings, and for the promise of rest there.
Could be that when you hear the word philanthropist, what pops into your head is a stodgy old One Percenter writing a big check to a dusty museum in exchange for his name on a brass plate in the lobby. Well, time to think again. At The Frederick Giving Project, the philanthropists are young. They?re not (necessarily) wealthy, and they?re too busy having snowball fights, kickball games and happy hours to even consider being stuffy. Oh yeah ? they?re also making a real difference in the Frederick community through both their financial largesse and volunteerism. Not a brass plate in sight.
Here?s how it works: $100 makes you a member of The Frederick Giving Project (i.e., a ?Giver?). The Giving Project pools all Giver contributions into an account at the Community Foundation of Frederick County, the tax-exempt umbrella organization that manages assets for a variety of local charitable funds. Then, once a year, Givers vote to choose a local nonprofit as their beneficiary (?Grantee?). Givers also commit to some volunteer work for that nonprofit during the next calendar year.
The concept, known as a ?giving circle,? is becoming an increasingly popular way for like-minded people of modest means to make an impact in causes they care about. Although there?s no age requirement, The Frederick Giving Project targets young professionals connected to the Frederick community.
?We say you have to be young at heart,? explains Rebecca Southers, chair of the group (or, according to its website, ?Empress?). ?If our events appeal to you ? our happy hours and general silliness, everything from a community-wide snowball fight to a supper club ? you?re our target audience. We do tend to be irreverent in our philanthropy. If you want to give back to your community playfully, you might want to check us out.?
Southers founded The Frederick Giving Project with an old friend from Hood College, Melissa Sines, with whom she reconnected at a nonprofit seminar. ?We were both sitting in a workshop about giving circles, talking about them theoretically. It sounded like what I was searching for, and I was so excited about it,? Southers says.
At one point, she saw Sines on the other side of the room raise her hand to ask a question, prefacing it with: ?I?ve heard a lot about giving circles and I?ve always wanted to start one ?.?
That?s when lightning struck for Southers. ?I looked at her and thought, ?We are doing this.?? Southers tracked down Sines afterward, and within the month, the two were planning how to set up a giving circle in Frederick, and they were pulling in other people.
That was March 2009. Southers and Sines, now the group?s vice chair (?Organizer Extraordinaire?), set up a fund at the Community Foundation and threw their launch party in December 2009. The two gathered members and donations throughout 2010, and by that December, the group?s Givers awarded $8,000 to Cakes for Cause. In 2011, the group volunteered at Cakes for Cause, which provides job and life skills training to youth aging out of foster care through internships at the Moxie Bakery & Caf? on North Market Street. They also staffed its farmer?s market stand and hosted happy hours and supper clubs at Moxie to raise funds for their grantee.
?Generally what the Giving Project does is make philanthropy accessible for younger people,? says Cakes for Cause Executive Director Elin Ross. ?It gives young people who may not have been exposed to philanthropy an opportunity to figure out what fits for them.?
Since the young professionals running the Giving Project come from the nonprofit world and are deeply familiar with the travails of grant writing, the group strives to make it as easy as possible for a nonprofit to become a Grantee. They start by soliciting a one-page letter of inquiry, then winnow them down to five finalists who are invited to submit a detailed proposal. Abbreviated versions are sent to Givers via an email survey. Each Giver gets one vote, no matter how much he or she has donated.
Out of 27 applicants last year, the group voted to make the Frederick Arts Council their 2012 grantee, donating more than $8,000 and volunteering in any capacity, including staffing the Frederick Festival of the Arts.
?Working with the Giving Project is pretty exceptional,? says Susie Miller, the arts council?s acting executive director. ?Not only do they provide funds, which are greatly appreciated, but more so, they volunteer their time and their expertise. That, for a nonprofit organization, is huge.? Miller notes that the Giving Project?s up-and-coming philanthropists? willingness to commit their time and money to a local nonprofit is one aspect that sets this giving circle apart from other organizations. ?It?s a great partnership. They?re using their knowledge and enthusiasm to help not just this nonprofit, but all the nonprofits they work with,? she says.
Southers believes The Frederick Giving Project serves the Frederick community in another way too. ?People in our generation want to feel connected,? she says. ?We commute long distances and converse virtually. There?s a common feeling that we?ve lost connection, and a deep need to reestablish it.? Staying connected to a giving circle fulfills that desire for community, Southers says. ?Just donating to a charity is so abstract. This is a social network of people who care and want to help. It?s a way to build friendships with other giving people.?
For more information about The Frederick Giving Project, its events and ways to get involved, visit www.frederickgivingproject.org.
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Public release date: 31-Jul-2012
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That was March 2009. Southers and Sines, now the group?s vice chair (?Organizer Extraordinaire?), set up a fund at the Community Foundation and threw their launch party in December 2009. The two gathered members and donations throughout 2010, and by that December, the group?s Givers awarded $8,000 to Cakes for Cause. In 2011, the group volunteered at Cakes for Cause, which provides job and life skills training to youth aging out of foster care through internships at the Moxie Bakery & Caf? on North Market Street. They also staffed its farmer?s market stand and hosted happy hours and supper clubs at Moxie to raise funds for their grantee.